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CAS RN: | 產品號碼: A2583
Anti-Sialyl Lewis A Monoclonal Antibody (1H4, Culture Supernatant)
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補充產品訊息:
Product Information
Volume: 0.2 mL
Product Form: Hybridoma culture supernatant (containing 10% fetal bovine serum)
Isotype : Mouse monoclonal antibody/IgG3, κ-chain
Preservative: 0.05% sodium azide
Stabilizer: None
Immunogen: Immunized with human colon cancer cells (Colo201), and screened using sialyl Lewis A-active glycolipids
Specificity: Sialyl Lewis A
Clone Name: 1H4
Applications
Immunohistochemistry,flow-cytometric analysis, ELISA, TLC-immunostaining, Western blotting, cell adhesion inhibition experiments.
Storage
Store in freezer (-20ﾟC to -80ﾟC). It is not recommended storing at 4ﾟC. Avoid repeated freezing and thawing.
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| 產品號碼 | A2583 |
| 外觀與形狀（20°C） | Liquid |
儲存條件 
|
Frozen (-80°C) |
| 應避免的情況 | Heat Sensitive |
產品規格
| Appearance | Orange to Red clear liquid |
| Titer(ELISA) | to pass test |
性質
GHS
相關法規
運輸資料
| HS編碼* | 3504.00-000 |
Application
Anti-Sialyl Lewis A Monoclonal Antibody (1H4, Culture Supernatant)
References
- Generation of two murine monoclonal antibodies that can discriminate N-glycolyl and N-acetyl neuraminic acid residues of GM2 gangliosides
- M. Miyake, M. Ito, S. Hitomi, S. Ikeda, T. Taki, M. Kurata, A. Hino, N. Miyake , R. Kannagi, Cancer Res. 1988, 48, 6154-6160.
- The abnormal occurrence and the differentiation-dependent distribution of N-acetyl and N-glycolyl species of the ganglioside GM2 in human germ cell tumors. A study with specific monoclonal antibodies
- Hypoxic culture induces expression of sialin, a sialic acid transporter, and cancer-associated gangliosides containing non-human sialic acid on human cancer cells
- Neuronal accumulation of α- and β-synucleins in the brain of a GM2 gangliosidosis mouse model
- K. Suzuki, E. Iseki, O. Katsuse, A. Yamaguchi, K. Katsuyama, I. Aoki, S. Yamanaka, K. Kosaka, Neuroreport 2003, 14, 551-554.
- β-hexosaminidase lentiviral vectors: transfer into the CNS via systemic administration
- The trigeminal retrograde transfer pathway in the treatment of neurodegeneration
- Conditional expression of human β-hexosaminidase in the neurons of Sandhoff disease rescues mice from neurodegeneration but not neuroinflammation
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